Gary A. Oltmans Associate Professor Ph.D., 1973, Univeristy Of Iowa Phone:847-578-8676 Office:HSB 2.612 Email:Gary.Oltmans@rosalindfranklin.edu

Research Interests

Neurotransmitter and receptor abnormalities underlying a genetic movement disorder; genetic and lesion-induced movement disorders.

Selected Publications

Litwak, J., Mercugliano, M., Chesselet, M.-F., and Oltmans, G.A. (1990) Increased glutamic acid decarboxylase (GAD) mRNA and GAD activity in cerebellar Purkinje cells following lesion-induced increases in cell firing Neurosci. Lett. 116:179-183.

Beales, M., Lorden, J.F., Walz, E. and Oltmans, G.A. (1990) Quantitative autoradiography reveals selective changes in cerebellar GABA receptors of the rat mutant dystonic J. Neuroscience 10:1874-1885.

Lutes, J., Lorden, J.F., Davis, B.J., and Oltmans, G.A. (1992) GABA levels and GAD immunoreactivity in the deep cerebellar nuclei of rats with altered olivo-cerebellar function Brain Res. Bull. 29:329-336.

Drengler, S.M. and Oltmans, G.A. (1993) Rapid increases in cerebellar Purkinje cell glutamic acid decarboxylase (GAD67) mRNA after lesion-induced increases in cell firing Brain Research 615:175-179.

Drengler, S.M., Lorden, J.F., Billitz, M.S., and Oltmans, G.A. (1996) Adrenergic Agents inhibit rapid increases in cerebellar Purkinje cell glutamic acid decarboxylase (GAD67) mRNA levels following climbing fiber lesions or reserpine treatment. J. Neuroscience 16:1844-1851.